Archive 1 Archive2

Type 0

Type zero diabetes is caused by Listeria chronic infection and causes a perpetual pre-diabetes state and fatigue and crash after eating. This is helped by getting enough sleep, but often this condition is coupled with insomnia. Rest often helps this type of person, and sleep deprivation makes the insomnia worse not better because the Listeria overgrows in sleep deprivation. The cause of this is because granulocytes get affected in sleep deprivation[1] and these cells are invaded by listeria.[2] . Excercise can also make it worse by reducing cellular immunity which is countered with beta glucan. [3] Listeria is fought with Ellagic Acid.

The body shuts down/mutates certain genes like listr2 and others during chronic and multigenerational listeria infection. Doing this limits the severity of Listeria infection, but at the same time causes diabetes. Some of these genes actually reduce diabetes when mutated so that is why often times this form of diabetes never progresses past prediabetes and is thus not clinically defined. Having type zero can enhance the other 3 types citation needed.

Diabetes enhances resistance to listeria, and this is likely due to fact that they were already infected which caused the diabetes.[4]

B6 mice are a mouse model for type 0 and are likely chronically and heritably infected with listeria and develop genetic changes that both make them more resistant to Listeria and at the same time more diabetic.[5]

Enhanced resistance to listeria gained with CD18 knockout and enhanced suseptibility to streptococcus at the same time[6]

Possible magnesium deficiency [7]

Type 1

Type one diabetes is caused by Mycobacterium autoimmunity on pancreatic cells that produce insulin. This causes a lack of insulin produced and can happen at any age. The patient produces antibodies to certain mycobacterium protiens; which antibodies attack zinc transporters in the pancreatic b cells. Mycobacterium is fought with garlic.

In addition to mycobacterium, toxoplasmosis can also cause destruction of pancreatic beta cells causing type 1 diabetes.

NOD mice are likely type 1 diabetic from generational mycobacterium infection.

Type 1 related to TB infection [8]

Mycobacterium likely trigger autoimmunity causing type 1 diabetes [9]

Mycobacterium associated with type 1 but not type 2 so is very likely the cause of the disease and not a result of high blood sugar. [10]

Mechanism for induction of type 1 diabetes by mycobacterium, antibodies to the bacteria protien are cross reactive with protiens in pancreatic beta cells, causing autoimmunity. [11]

Another mechanism is likely that chronic mycobacterium infection leads the body to shut down retinoids production to fend off mycobacteria and thus doing causes pancreatic beta cell trouble. This is based on Vitamin A causes problems with pancreatic beta cells and type 1 diabetes [12] [13] and also people have said that vitamin a can cause high cholesterol which was cured with garlic (which treats mycobacterium).

Alpha cells can convert to beta cells [14] and possibly red light therapy [15].

Can also be from mumps or rubella [16]

Type 2

Type two diabetes is caused by Haemophilus bacteria and causes the cells to loose sensitivity to insulin. This is because haemophilus uses nicotinamide riboside to grow [17], causing deficiency. Treating the patient with nicotinamide (flush free niacin) will help [18] although it will likely cause overgrowth of the bacteria since it is a precursor to nicotinamide riboside. Cinnamon capsules or essential oil will help control the bacteria or anything else that neutralizes haemophilus. This is why cinnamon is effective in treating type 2 diabetes. [19]

BLK mouse model likely is chronically and heritably infected with haemophilus. [20]

Clove extract may help [21]

Possible magnesium deficiency [22]

Type 3

Cognitive Decline is considered Type 3 diabetes [23] and is related to a Folic Acid deficiency. This is likely caused by pseudomonas which degrades folic acid.[24] Pseudomonas is treated with cinnamon just like haemophilus in type 2.


Phosphate diabetes is common in regular diabetes and results in phosphorus, potassium, and magnesium deficiency [25]

Quick sodium repletion can lead to demyelination [26]

Insulin drives potassium into cells [27]

Low phosphate causes elevated insu!in secretion and thus probably insulin resistance [28]

Other pages that link to Diabetes:


Attachments to Diabetes:

Password to edit: nature